Caspases and p38 MAPK regulate endothelial cell adhesiveness for mesenchymal stem cells

Authors:
Potapova IA, Cohen IS, Doronin SV
In:
Source: PLoS ONE
Publication Date: (2013)
Issue: 8(9): e73929
Research Area:
Stem Cells
Basic Research
Cells used in publication:
Endothelial, umbilical vein, human (HUVEC)
Species: human
Tissue Origin: vein
Mesenchymal stem cell (MSC), human
Species: human
Tissue Origin: bone marrow
Abstract
Mesenchymal stem cells natively circulating or delivered into the blood stream home to sites of injury. The mechanism of mesenchymal stem cell homing to sites of injury is poorly understood. We have shown that the development of apoptosis in endothelial cells stimulates endothelial cell adhesiveness for mesenchymal stem cells. Adhesion of mesenchymal stem cells to apoptotic endothelial cells depends on the activation of endothelial caspases and p38 MAPK. Activation of p38 MAPK in endothelial cells has a primary effect while the activation of caspases potentiates the mesenchymal stem cell adhesion. Overall, our study of the mesenchymal stem cell interaction with endothelial cells indicates that mesenchymal stem cells recognize and specifically adhere to distressed/apoptotic endothelial cells.